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Bernhard Hennig, Ph.D., R.D.
Director, University of Kentucky Superfund Basic Research Program
Professor of Nutrition and Toxicology

591 CT Wethington Building
900 South Limestone
Lexington, KY 40536-0200
Tel: (859)-323-4933
Fax: (859)-257-1811
e-mail: bhennig@uky.edu

Academic Appointments:

• College of Agriculture
• Graduate Center for Nutritional Sciences
• Graduate Center for Toxicology
• Center of Membrane Sciences

Education:

• B.A., Biochemistry, San Francisco State University, San Francisco, CA, 1973-1977
• M.S., Nutrition, Colorado State University, Fort Collins, CO, 1977-1979.
• Ph.D., Nutrition, Iowa State University, Ames, IA, 1979-1982.
• Postdoctoral, Cardiovascular Center, College of Medicine, Department of    Biochemistry, University of Iowa, Iowa City, IA., 1983-1984
• Postdoctoral Research Fellowship, HL 07121, National Heart Lung and Blood    Institute, NIH, Research Director: Dr. Arthur A. Spector)

Awards:

• Borden Award for Significant Research in Nutrition
• Clintec Award for Excellence in the Science of Nutrition
• Grace A. Goldsmith award for Significant Current Achievements in Nutrition

Research:

Dr. Hennig’s research emphasis involves the utilization of tissue culture model systems in the study of nutrition and atherosclerosis, with emphasis in the role of nutrients on biomedical and molecular mechanisms of vascular endothelial cell function, injury and protection. His research has focused on the injurious effects of individual fats (e.g., fatty acids, cholesterol and their oxidation derivatives) to vascular endothelial cells and protection against such injury by certain vitamins and minerals (e.g., vitamin E and zinc). Because the nutritional environment of the vascular endothelium can influence cytokine-mediated endothelial activation or dysfunction, vascular effects of specific environmental contaminants, such as PCBs, in relation to endolethial cell activation and the pathology of atherosclerosis. Results from these studies may suggest novel therapeutic interventions by further understanding “endothelial cell nutrition” as a key variable in the etiology of diseases such as atherosclerosis.